content/projects/a01/_index.md

@@ -1,20 +1,26 @@
 ---
-title: "A01: The neural code of stimulus-triggered territorial aggression"
+title: 'A01: The neural code of stimulus-triggered territorial aggression'
 contributors:
-- Marc Spehr
-- lena terlau
+- lena-terlau
+- marc-spehr
 sites:
-- Aachen
+- aachen
 topics:
 - research-area-a
 - dfg-206-02
 weight: 1000
 ---
 
-{{< lead >}}
-A unique experimental model will be used to study the neurobiological basis of aggressive animal behavior and translate novel findings about its cellular and circuit underpinnings into human phenotypes of pathological aggression.
-Specifically, the post-weaning social isolation model of early-life adverse experience allows experimental access to dissect the ‘switch’ from functional adaptive aggression to excessive pathological aggressive behavior.
-In male mice, conspecific chemostimuli trigger innate aggressive behavior.
-The relevant aggression-promoting circuits along the(sensory) input to (aggressive) output axis comprise the accessory olfactory bulb (AOB), a hub for processing of social chemosignals, and the medial amygdala (MeA), a crucial control centre for regulation of aggressive behavior.
-Therefore, this project addresses the principles that govern aggression-promoting information transfer along the AOB-to-MeA signaling pathway.
-{{< /lead >}}
+A unique experimental model will be used to study the neurobiological basis of
+aggressive animal behavior and translate novel findings about its cellular and
+circuit underpinnings into human phenotypes of pathological aggression.
+Specifically, the post-weaning social isolation model of early-life adverse
+experience allows experimental access to dissect the ‘switch’ from functional
+adaptive aggression to excessive pathological aggressive behavior. In male mice,
+conspecific chemostimuli trigger innate aggressive behavior. The relevant
+aggression-promoting circuits along the(sensory) input to (aggressive) output
+axis comprise the accessory olfactory bulb (AOB), a hub for processing of social
+chemosignals, and the medial amygdala (MeA), a crucial control centre for
+regulation of aggressive behavior. Therefore, this project addresses the
+principles that govern aggression-promoting information transfer along the AOB-
+to-MeA signaling pathway.

content/projects/a02/_index.md

@@ -1,28 +1,30 @@
 ---
-title: "A02: Context effects on threat processing in dependence of testosterone levels"
+title: 'A02: Context effects on threat processing in dependence of testosterone levels'
 contributors:
-- Katja Bertsch
-- Sabine Herpertz
-- Ute Habel
-- Isabel Neumann
-- Nick Worm
+- isabel-neumann
+- katja-bertsch
+- nick-worm
+- sabine-herpertz
+- ute-habel
 sites:
-- wuerzburg
-- heidelberg
 - aachen
+- heidelberg
+- wuerzburg
 topics:
-- research-area-a
 - dfg-206-09
 - dfg-206-10
+- research-area-a
 weight: 1010
 ---
 
-{{< lead >}}
-The focus will be on the influences of a provocative context on social threat processing in AMD under different testosterone
-levels. Specifically, the project aims to analyze the modulating function of context under testosterone application versus
-suppression on threat sensitivity in healthy controls as well as patient groups. Additionally, we will determine the
-influence of endogenous hormone variations (testosterone, oxytocin, estrogen and cortisol) on NVS in high versus low
-aggressive patients in a large group of patients recruited in Q01. With this sample, we will try to identify multidimensional
-biosignatures based on hormonal levels in combination with fMRI measures of amygdala and amygdala-prefrontal
-connectivity, NVS measures by questionnaires, aggression measures and psychopathological data.
-{{< /lead >}}
+The focus will be on the influences of a provocative context on social threat
+processing in AMD under different testosterone levels. Specifically, the project
+aims to analyze the modulating function of context under testosterone
+application versus suppression on threat sensitivity in healthy controls as well
+as patient groups. Additionally, we will determine the influence of endogenous
+hormone variations (testosterone, oxytocin, estrogen and cortisol) on NVS in
+high versus low aggressive patients in a large group of patients recruited in
+Q01. With this sample, we will try to identify multidimensional biosignatures
+based on hormonal levels in combination with fMRI measures of amygdala and
+amygdala-prefrontal connectivity, NVS measures by questionnaires, aggression
+measures and psychopathological data.

content/projects/a03/_index.md

@@ -1,17 +1,18 @@
 ---
-title: "A03: Modulation of aggression by acute threat"
+title: 'A03: Modulation of aggression by acute threat'
 contributors:
-- Gabriele Ende
-- Christian Schmahl
+- christian-schmahl
+- gabriele-ende
+- milad-amini-masouleh
+- neha-vats
 sites:
 - mannheim
 topics:
-- research-area-a
 - dfg-206-10
+- research-area-a
 weight: 1020
 ---
 
-{{< lead >}}
 The neural and neurochemical patterns of acute threat as modulators of
 aggression in BPD will be investigated in this project. The modulation of
 aggressive responses under acute threat is induced by the threat-of-shock
@@ -20,9 +21,8 @@ responses are modulated by threat, and which neurofunctional and neurochemical
 patterns underlie these responses during safe and threat conditions. MR
 spectroscopy will be used in patients to assess glutamate and GABA levels. In a
 further translational approach, the least and the most aggressive/impulsive
-recombinant inbred mouse lines identified in [C01 in Frankfurt]({{< relref
-"/projects/c01" >}}) will be tested in Mannheim with animal MR spectroscopy at
-9.4T to determine the relationship between glutamate, GABA, impulsivity, and
-aggression in these mouse lines as well as in comparable brain regions
-assessing neurofunctional and neurochemical patterns.
-{{< /lead >}}
+recombinant inbred mouse lines identified in C01 in Frankfurt will be tested in
+Mannheim with animal MR spectroscopy at 9.4T to determine the relationship
+between glutamate, GABA, impulsivity, and aggression in these mouse lines as
+well as in comparable brain regions assessing neurofunctional and neurochemical
+patterns.

content/projects/a04/_index.md

@@ -1,22 +1,25 @@
 ---
-title: "A04:  Implicit chemosensory threat signals as stimulators of amygdala hyperresponsiveness in AMD"
+title: 'A04: Implicit chemosensory threat signals as stimulators of amygdala hyperresponsiveness
+  in AMD'
 contributors:
-- Ute Habel
-- Natalia Chechko
+- christoph-mallmann
+- natalia-chechko
+- ute-habel
 sites:
 - aachen
 topics:
-- research-area-a
 - dfg-206-09
 - dfg-206-10
+- research-area-a
 weight: 1030
 ---
 
-{{< lead >}}
-We make use of threat-related chemosensory stimuli, namely body odor, acquired during aggressive behavior (boxing)
-and unconsciously perceived, to investigate heightened amygdala responses to threat stimuli in aggressive patients. Body
-odors have the major advantage of being directly projected into the amygdala, circumventing cortical preprocessing,
-thereby enabling the differentiation of mechanisms between bottom-up altered limbic processing and top-down modulated
-altered cognitive evaluation. We investigate the potential of such body odors to bias responses to ambiguous visual social
-cues towards threat and their effects during peripersonal space (PPS) violation where they may be especially relevant.
-{{< /lead >}}
+We make use of threat-related chemosensory stimuli, namely body odor, acquired
+during aggressive behavior (boxing) and unconsciously perceived, to investigate
+heightened amygdala responses to threat stimuli in aggressive patients. Body
+odors have the major advantage of being directly projected into the amygdala,
+circumventing cortical preprocessing, thereby enabling the differentiation of
+mechanisms between bottom-up altered limbic processing and top-down modulated
+altered cognitive evaluation. We investigate the potential of such body odors to
+bias responses to ambiguous visual social cues towards threat and their effects
+during peripersonal space (PPS) violation where they may be especially relevant.

content/projects/a05/_index.md

@@ -1,22 +1,25 @@
 ---
-title: "A05: Peripersonal space violations and social threat: daily-life psychological and neural mechanisms of environmental risk for reactive aggression"
+title: 'A05: Peripersonal space violations and social threat: daily-life psychological
+  and neural mechanisms of environmental risk for reactive aggression'
 contributors:
-- Heike Tost
-- Andreas Meyer-Lindenberg
+- alper-koelgesiz
+- andreas-meyer-lindenberg
+- habiba-hassan
+- heike-tost
 sites:
 - mannheim
 topics:
-- research-area-a
-- dfg-206-08
 - dfg-206-09
+- research-area-a
 weight: 1040
 ---
 
-{{< lead >}}
-Peripersonal space, the representation of the space immediately surrounding the body, will be studied as an underlying factor
-for threat experience. Early-life stressors and daily-life stressors will be tested as factors influencing PPS processing
-and associated specific brain activation patterns. Location tracking and geoinformatics mapping, virtual reality (VR)
-experiments, physiological stress markers, and brain function during the processing of PPS violations in healthy at-
-risk individuals will be used to identify predictive biomarkers related to psychiatric risk, enhanced neural behavioral
-sensitivity to PPS interference and reactive aggression in daily life.
-{{< /lead >}}
+Peripersonal space, the representation of the space immediately surrounding the
+body, will be studied as an underlying factor for threat experience. Early-life
+stressors and daily-life stressors will be tested as factors influencing PPS
+processing and associated specific brain activation patterns. Location tracking
+and geoinformatics mapping, virtual reality (VR) experiments, physiological
+stress markers, and brain function during the processing of PPS violations in
+healthy at- risk individuals will be used to identify predictive biomarkers
+related to psychiatric risk, enhanced neural behavioral sensitivity to PPS
+interference and reactive aggression in daily life.

content/projects/a06/_index.md

@@ -1,26 +1,31 @@
 ---
-title: "A06: Decoding dynamic reciprocal neural mechanism underlying reactive aggression: Insights from fMRI and fNIRS hyperscanning"
+title: 'A06: Decoding dynamic reciprocal neural mechanism underlying reactive aggression:
+  Insights from fMRI and fNIRS hyperscanning'
 contributors:
-- Kerstin Konrad
-- Andreas Meyer-Lindenberg
-- Vanessa Reindl
+- andreas-meyer-lindenberg
+- kerstin-konrad
+- mina-misic
+- neele-ulken
+- vanessa-reindl
 sites:
 - aachen
 - mannheim
 topics:
-- research-area-a
 - dfg-206-10
+- research-area-a
 weight: 1050
 ---
 
-{{< lead >}}
-The project employs fMRI and functional near-infrared
-spectroscopy (fNIRS) hyperscanning techniques to explore how brain-to-brain synchrony and dynamic processes within
-peer dyads facilitate or inhibit aggressive behavior under diverse levels of provocation in adolescent patients and controls.
-In two fully interactive tasks, we will probe aggressive behavior towards a task partner, and quantify the building of
-interpersonal trust/distrust applying a social interaction and economic exchange paradigm. These paradigms will be
-employed within dyads in fMRI hyperscanning settings and extended by group-based fNIRS methods in triads to study
-effects of peers, social exclusion, and coalitions on aggressive behavior in semi-naturalistic interactions. Between-brain
-neural synchrony will be computed and related to everyday social experiences and individual predispositions to identify
-markers for the prediction of aggressive behavior.
-{{< /lead >}}
+The project employs fMRI and functional near-infrared spectroscopy (fNIRS)
+hyperscanning techniques to explore how brain-to-brain synchrony and dynamic
+processes within peer dyads facilitate or inhibit aggressive behavior under
+diverse levels of provocation in adolescent patients and controls. In two fully
+interactive tasks, we will probe aggressive behavior towards a task partner, and
+quantify the building of interpersonal trust/distrust applying a social
+interaction and economic exchange paradigm. These paradigms will be employed
+within dyads in fMRI hyperscanning settings and extended by group-based fNIRS
+methods in triads to study effects of peers, social exclusion, and coalitions on
+aggressive behavior in semi-naturalistic interactions. Between-brain neural
+synchrony will be computed and related to everyday social experiences and
+individual predispositions to identify markers for the prediction of aggressive
+behavior.

content/projects/a07/_index.md

@@ -1,23 +1,28 @@
 ---
-title: "A07: The intestinal microbiota as a regulator of aggressive and impulsive behavior"
+title: 'A07: The intestinal microbiota as a regulator of aggressive and impulsive
+  behavior'
 contributors:
-- David Slattery
-- Andreas Reif
+- andreas-reif
+- antonia-fritsch
+- david-slattery
+- ina-kuschel
+- seyedali-hashemi
 sites:
 - frankfurt
 topics:
-- research-area-a
 - dfg-206-09
+- research-area-a
 weight: 1060
 ---
 
-{{< lead >}}
-This translational project
-investigates sex-dependent behavioral effects of faecal microbiota transplantation to microbiome-depleted mice
-from AMD patients (selected based on their aggressive and impulsive traits from [Q01]({{< relref "/projects/q01" >}})), as well as healthy controls.
-Impulsivity will be assessed via the continuous performance test and responses towards acute threat via the escalated
-resident intruder test. The goal is to determine the sex-dependent effects of faecal transplantation on selected readouts
-involved in the transfer of the patient’s phenotype to the mice, such as immune parameters, sex hormones, neuronal
-activity (and morphology, e.g., neurite outgrowth, spines, etc.), and gene expression (e.g., Rbfox1 from prior studies and
-novel candidates from [C01]({{< relref "/projects/c01" >}}) and [C04]({{< relref "/projects/c04" >}})).
-{{< /lead >}}
+This translational project investigates sex-dependent behavioral effects of
+faecal microbiota transplantation to microbiome-depleted mice from AMD patients
+(selected based on their aggressive and impulsive traits from Q01), as well as
+healthy controls. Impulsivity will be assessed via the continuous performance
+test and responses towards acute threat via the escalated resident intruder
+test. The goal is to determine the sex-dependent effects of faecal
+transplantation on selected readouts involved in the transfer of the patient’s
+phenotype to the mice, such as immune parameters, sex hormones, neuronal
+activity (and morphology, e.g., neurite outgrowth, spines, etc.), and gene
+expression (e.g., Rbfox1 from prior studies and novel candidates from C01 and
+C04).

content/projects/a08/_index.md

@@ -1,29 +1,32 @@
 ---
-title: "A08: The metabolic lung-brain axis in aggressive behavior in patients with AMD"
+title: 'A08: The metabolic lung-brain axis in aggressive behavior in patients with
+  AMD'
 contributors:
-- Thomas Frodl
-- David Slattery
-- Gabriele Ende
+- david-slattery
+- gabriele-ende
+- thomas-frodl
 sites:
 - aachen
 - frankfurt
 - mannheim
 topics:
-- research-area-a
 - dfg-206-09
 - dfg-206-10
+- research-area-a
 weight: 1070
 ---
 
-{{< lead >}}
-Beta-hydroxy-butyrate (BHB), a ketone body, is negatively associated with aggressive behavior. BHB is a metabolite and an
-active signaling substrate involved in epigenetic regulation of e.g., neurotrophic factor genes in the brain. Of the three
-main ketone bodies, acetone, acetoacetate and BHB, acetone is a very volatile compound, mainly eliminated through
-respiration, thus can be measured non-invasively in breath. A reduction of acetone in breath has been found to highly
-correlate with BHB in blood and be associated with symptom severity in schizophrenia (Jiang et al. 2022). Using MR
-spectroscopy, A08 aims to (1) identify whether acetone and other volatile organic compounds in breath are associated
-with aggression and acute threat processing in mental disorders and (2) to examine whether these breath markers are
-associated with direct metabolic brain correlates (like BHB, glutamate) and with the brain-derived neurotrophic factor
-(BDNF) levels in plasma. In a translational approach, (3) we will test if supplementation of BHB reduces aggressive
-behavior in mice.
-{{< /lead >}}
+Beta-hydroxy-butyrate (BHB), a ketone body, is negatively associated with
+aggressive behavior. BHB is a metabolite and an active signaling substrate
+involved in epigenetic regulation of e.g., neurotrophic factor genes in the
+brain. Of the three main ketone bodies, acetone, acetoacetate and BHB, acetone
+is a very volatile compound, mainly eliminated through respiration, thus can be
+measured non-invasively in breath. A reduction of acetone in breath has been
+found to highly correlate with BHB in blood and be associated with symptom
+severity in schizophrenia (Jiang et al. 2022). Using MR spectroscopy, A08 aims
+to (1) identify whether acetone and other volatile organic compounds in breath
+are associated with aggression and acute threat processing in mental disorders
+and (2) to examine whether these breath markers are associated with direct
+metabolic brain correlates (like BHB, glutamate) and with the brain-derived
+neurotrophic factor (BDNF) levels in plasma. In a translational approach, (3) we
+will test if supplementation of BHB reduces aggressive behavior in mice.

content/projects/b01/_index.md

@@ -1,25 +1,31 @@
 ---
-title: "B01:  Neurobehavioral effects of repetitive prefrontal transcranial direct current stimulation (tDCS) on pathological aggression"
+title: 'B01: Neurobehavioral effects of repetitive prefrontal transcranial direct
+  current stimulation (tDCS) on pathological aggression'
 contributors:
-- Carmen Weidler
-- Andreas Reif
+- andreas-reif
+- antonia-fritsch
+- carmen-weidler
+- ina-kuschel
+- luca-lasogga
 sites:
-- Aachen
-- Frankfurt
+- aachen
+- frankfurt
 topics:
-- research-area-b
 - dfg-206-09
+- research-area-b
 - dfg-206-10
 weight: 2000
 ---
 
-{{< lead >}}
-TDCS will be used as an interventional tool to decrease aggression. Using a simultaneous
-tDCS – fMRI approach, the project aims to enhance cognitive control by repeated prefrontal brain stimulation, investigating
-its effect on aggression. In addition to gauging tDCS responsivity, identifying the role of individual factors such as genetic
-profiles in aggression will be a particular focus of this project. By examining brain activity at multiple time points (e.g.,
-before, during multiple stimulation sessions and after tDCS), it will add to the understanding of mechanisms underlying
-neural tDCS effects and help to identify individual factors that predict responsiveness to the stimulation. To determine
-the therapeutic potential, we will include psychiatric patients with substance use problems, a group of criminal, violent
-offenders, and healthy matched controls.
-{{< /lead >}}
+TDCS will be used as an interventional tool to decrease aggression. Using a
+simultaneous tDCS – fMRI approach, the project aims to enhance cognitive control
+by repeated prefrontal brain stimulation, investigating its effect on
+aggression. In addition to gauging tDCS responsivity, identifying the role of
+individual factors such as genetic profiles in aggression will be a particular
+focus of this project. By examining brain activity at multiple time points
+(e.g., before, during multiple stimulation sessions and after tDCS), it will add
+to the understanding of mechanisms underlying neural tDCS effects and help to
+identify individual factors that predict responsiveness to the stimulation. To
+determine the therapeutic potential, we will include psychiatric patients with
+substance use problems, a group of criminal, violent offenders, and healthy
+matched controls.

content/projects/b02/_index.md

@@ -1,26 +1,30 @@
 ---
-title: "B02: Young offenders’ self-regulation deficit as a common mechanism for aggressive behavior and psychopathology - neural mechanisms and role of adverse childhood experiences"
+title: 'B02: Young offenders’ self-regulation deficit as a common mechanism for aggressive
+  behavior and psychopathology - neural mechanisms and role of adverse childhood experiences'
 contributors:
-- Wolfgang Retz
-- Oliver Tüscher
+- alexandra-sebastian
+- oliver-tuescher
+- wolfgang-retz
 sites:
 - mainz
 topics:
-- research-area-b
-- dfg-206-04
 - dfg-206-09
+- research-area-b
 - dfg-206-10
+- dfg-206-04
 weight: 2010
 ---
 
-{{< lead >}}
-This project aims to identify
-cognitive and emotion control deficits in the context of negative valence and threat interference and their association
-with ACE in young offenders. Complementary to other projects, this project will focus on a group of young people
-defined by their propensity to aggression showing at the same time more severe psychopathologies. In a series of studies
-using multimodal imaging (EEG-fMRI, EEG-sMRI) in combination with naturalistic longitudinal follow-up (ecological
-momentary assessment (EMA)) B02 will identify the neural mechanisms and predictors of self-regulation deficits as a
-putative common developmental pathway for both, aggressive behavior, and psychopathology. Additionally, B02 will
-seek to causally confirm neural network mechanisms of inhibitory control and emotion regulation deficits as the basis of
-aggressive behavior and associated psychopathology by real-time EEG-triggered TMS-stimulation in young offenders.
-{{< /lead >}}
+This project aims to identify cognitive and emotion control deficits in the
+context of negative valence and threat interference and their association with
+ACE in young offenders. Complementary to other projects, this project will focus
+on a group of young people defined by their propensity to aggression showing at
+the same time more severe psychopathologies. In a series of studies using
+multimodal imaging (EEG-fMRI, EEG-sMRI) in combination with naturalistic
+longitudinal follow-up (ecological momentary assessment (EMA)) B02 will identify
+the neural mechanisms and predictors of self-regulation deficits as a putative
+common developmental pathway for both, aggressive behavior, and psychopathology.
+Additionally, B02 will seek to causally confirm neural network mechanisms of
+inhibitory control and emotion regulation deficits as the basis of aggressive
+behavior and associated psychopathology by real-time EEG-triggered TMS-
+stimulation in young offenders.

content/projects/b03/_index.md

@@ -1,25 +1,27 @@
 ---
-title: "B03: A process-based brain-computer interface to modulate aggressive behavior – a real-time fMRI neurofeedback study"
+title: 'B03: A process-based brain-computer interface to modulate aggressive behavior
+  – a real-time fMRI neurofeedback study'
 contributors:
-- Klaus Mathiak
-- Christian Paret
-- Jana Zweerings
+- christian-paret
+- jana-zweerings
+- klaus-mathiak
 sites:
 - aachen
 - mannheim
 topics:
-- research-area-b
 - dfg-206-09
+- research-area-b
 weight: 2020
 ---
 
-{{< lead >}}
-Probe the self-regulation of CS networks in adults and adolescents
-diagnosed with mental disorders related to frequent stress-associated affective outbursts and aggressive symptoms
-in posttraumatic stress disorder (PTSD), and BPD. The patients will subsequently be trained to regulate the frontal
-control network to varying acute threat in a double-blind, randomized, controlled design. An immersive, virtual brain-
-computer-interface (BCI) will allow for a culture- and age-sensitive, personalized training approach. The aim of the present
-investigation is to assess feasibility of the approach according to four clinical markers: Reduction of perceived threat
-and aggressive behavior in daily life, improved control in the face of unfair provocation, and neurofeedback-specific
-modulation of the neural networks.
-{{< /lead >}}
+Probe the self-regulation of CS networks in adults and adolescents diagnosed
+with mental disorders related to frequent stress-associated affective outbursts
+and aggressive symptoms in posttraumatic stress disorder (PTSD), and BPD. The
+patients will subsequently be trained to regulate the frontal control network to
+varying acute threat in a double-blind, randomized, controlled design. An
+immersive, virtual brain- computer-interface (BCI) will allow for a culture- and
+age-sensitive, personalized training approach. The aim of the present
+investigation is to assess feasibility of the approach according to four
+clinical markers: Reduction of perceived threat and aggressive behavior in daily
+life, improved control in the face of unfair provocation, and neurofeedback-
+specific modulation of the neural networks.

content/projects/b04/_index.md

@@ -1,26 +1,30 @@
 ---
-title: "B04: Investigating psychological and neural correlates of intimate partner violence"
+title: 'B04: Investigating psychological and neural correlates of intimate partner
+  violence'
 contributors:
-- Lisa Wagels
-- Andreas Meyer-Lindenberg
-- Katja Bertsch
-- Julia Koch
-- Julia Schräder
-- Linda Wilkin-Krug
-
+- andreas-meyer-lindenberg
+- julia-schraeder
+- katja-bertsch
+- linda-wilkin-krug
+- lisa-wagels
 sites:
 - aachen
 - mannheim
 - wuerzburg
 topics:
-- research-area-b
 - dfg-206-09
+- research-area-b
 - dfg-206-10
 weight: 2030
 ---
 
-{{< lead >}}
-Focus on the neural correlates of characterizing cognitive control deficits during conflict situations.
-The project will investigate patients with varying levels of cognitive control along with their close partners (sibling or intimate partner) to identify the dynamics of self-regulation and co-regulation in provoked conflict situations in patients with control deficits. To identify the precursors and dynamics of conflict escalation, the project will apply measures of behavioral reactions, skin conductance, simulated or real conflict, fMRI and fMRI-hyperscanning techniques and physiological measures.
-Neuroimaging data will also be combined with information on stress, control and conflicts in real-life via EMA.
-{{< /lead >}}
+Focus on the neural correlates of characterizing cognitive control deficits
+during conflict situations. The project will investigate patients with varying
+levels of cognitive control along with their close partners (sibling or intimate
+partner) to identify the dynamics of self-regulation and co-regulation in
+provoked conflict situations in patients with control deficits. To identify the
+precursors and dynamics of conflict escalation, the project will apply measures
+of behavioral reactions, skin conductance, simulated or real conflict, fMRI and
+fMRI-hyperscanning techniques and physiological measures. Neuroimaging data will
+also be combined with information on stress, control and conflicts in real-life
+via EMA.

content/projects/b05/_index.md

@@ -1,8 +1,11 @@
 ---
-title: "B05: Predictors and (neuro-)biological correlates of (cyber-)bullying and victimization in real-life contexts"
+title: 'B05: Predictors and (neuro-)biological correlates of (cyber-)bullying and
+  victimization in real-life contexts'
 contributors:
-- Nathalie Holz
-- Tobias Banaschewski
+- anna-erdogan
+- nathalie-holz
+- nilakshi-vaidya
+- tobias-banaschewski
 sites:
 - mannheim
 topics:
@@ -11,12 +14,13 @@ topics:
 weight: 2040
 ---
 
-{{< lead >}}
-Focus on the investigation of a lack of cognitive control in bullies and victims that contributes to the
-risk of developing mental health problems. Therefore, the project will assess bullies and their victims in real-life and digital
-social interactions to investigate how aberrant cognitive and affective prefrontal control and sensitivity to peer rejection
-with accompanied alterations in autonomic arousal may increase externalizing and internalizing behavior. To this end,
-a unique combination of ambulatory assessments of (cyber-)bullying, functional neuroimaging (emotion regulation,
-inhibition, social exclusion), physiological assessments (heart rate variability) and clinical trait-related questionnaires
-will be applied. Decoding dynamic
-{{< /lead >}}
+Focus on the investigation of a lack of cognitive control in bullies and victims
+that contributes to the risk of developing mental health problems. Therefore,
+the project will assess bullies and their victims in real-life and digital
+social interactions to investigate how aberrant cognitive and affective
+prefrontal control and sensitivity to peer rejection with accompanied
+alterations in autonomic arousal may increase externalizing and internalizing
+behavior. To this end, a unique combination of ambulatory assessments of
+(cyber-)bullying, functional neuroimaging (emotion regulation, inhibition,
+social exclusion), physiological assessments (heart rate variability) and
+clinical trait-related questionnaires will be applied.

content/projects/c01/_index.md

@@ -1,23 +1,27 @@
 ---
-title: "C01: Gene-environment interactions and the role of impulsivity in responding to acute threats: early life stress and escalated aggression in recombinant inbred mouse strains"
+title: 'C01: Gene-environment interactions and the role of impulsivity in responding
+  to acute threats: early life stress and escalated aggression in recombinant inbred
+  mouse strains'
 contributors:
 - aet-oleary
-- david slattery
+- david-slattery
+- hande-betuel-oezsoy
 sites:
 - frankfurt
 topics:
-- research-area-c
 - dfg-206-09
+- research-area-c
 weight: 3000
 ---
 
-{{< lead >}}
-Sex-dependent effects
-and gene-environment interactions will be investigated by applying escalating aggression paradigms. Specifically, the
-project will investigate the effects of early life stress on aggression in response to threat and hyperactivity as well as social
-decision-making in 32 BXD mouse strains, the progenitor strains (C057Bl/6J and DBA/2J), and the F1 BXD cross. The project
-aims to identify the quantitative trait loci (QTL) and putative candidate genes contained within the QTL and associate
-them with specific behavioral responses of stressed and unstressed cohorts of mice. The publicly available database
-GeneNetwork (www.genenetwork.org) will be used to validate the findings which include measurements of mRNA and
-protein expression, and methylation patterns in mouse brains
-{{< /lead >}}
+Sex-dependent effects and gene-environment interactions will be investigated by
+applying escalating aggression paradigms. Specifically, the project will
+investigate the effects of early life stress on aggression in response to threat
+and hyperactivity as well as social decision-making in 32 BXD mouse strains, the
+progenitor strains (C057Bl/6J and DBA/2J), and the F1 BXD cross. The project
+aims to identify the quantitative trait loci (QTL) and putative candidate genes
+contained within the QTL and associate them with specific behavioral responses
+of stressed and unstressed cohorts of mice. The publicly available database
+GeneNetwork (www.genenetwork.org) will be used to validate the findings which
+include measurements of mRNA and protein expression, and methylation patterns in
+mouse brains.

content/projects/c02/_index.md

@@ -1,28 +1,31 @@
 ---
-title: "C02: Aggressive decisions in social conflicts: Neuro-cognitive models for healthy individuals and psychiatric patients with high scores of aggression"
+title: 'C02: Aggressive decisions in social conflicts: Neuro-cognitive models for
+  healthy individuals and psychiatric patients with high scores of aggression'
 contributors:
-- Christoph Korn
-- Klaus Mathiak
+- christoph-korn
+- klaus-mathiak
+- moritz-burghardt
 sites:
 - aachen
 - heidelberg
 topics:
-- research-area-c
-- dfg-110-02
-- dfg-206-04
-- dfg-206-08
 - dfg-206-09
+- research-area-c
+- dfg-206-04
+- dfg-110-02
 weight: 3010
 ---
 
-{{< lead >}}
-Develop virtual scenarios to assess decision strategies in cartoon-like and naturalistic
-contexts. The core question is how healthy individuals and patients make (mal-)adaptive aggressive decisions in social
-conflicts given their threat sensitivity, cognitive functions, and learning experience. We plan to present mathematically
-well-defined aggressive decision scenarios to healthy participants as well as patients across diagnostic categories with
-high scores of aggressive behavior, threat sensitivity, and inference of hostile intent in others. Computational models that
-accurately explain behavioral choices and neural responses (tested using fMRI and pupillometry) will be developed to
-identify the aggressive decision strategies humans employ in approach-avoidance conflicts of increasing complexity and
-ecological realism. The purpose will be to determine if patients use overly aggressive strategies that are not warranted by
-the necessary defense of self-threats and underlying neural circuits.
-{{< /lead >}}
+Develop virtual scenarios to assess decision strategies in cartoon-like and
+naturalistic contexts. The core question is how healthy individuals and patients
+make (mal-)adaptive aggressive decisions in social conflicts given their threat
+sensitivity, cognitive functions, and learning experience. We plan to present
+mathematically well-defined aggressive decision scenarios to healthy
+participants as well as patients across diagnostic categories with high scores
+of aggressive behavior, threat sensitivity, and inference of hostile intent in
+others. Computational models that accurately explain behavioral choices and
+neural responses (tested using fMRI and pupillometry) will be developed to
+identify the aggressive decision strategies humans employ in approach-avoidance
+conflicts of increasing complexity and ecological realism. The purpose will be
+to determine if patients use overly aggressive strategies that are not warranted
+by the necessary defense of self-threats and underlying neural circuits.

content/projects/c03/_index.md

@@ -1,26 +1,29 @@
 ---
-title: "C03: Distributed network control and interventions to frustrative non-reward and threat triggered aggressions"
+title: 'C03: Distributed network control and interventions to frustrative non-reward
+  and threat triggered aggressions'
 contributors:
-- Wolfgang Kelsch
-- Wolfgang Weber-Fahr
+- wolfgang-kelsch
+- wolfgang-weber-fahr
 sites:
 - mainz
 - mannheim
 topics:
-- research-area-c
 - dfg-206-09
+- research-area-c
 - dfg-206-10
 weight: 3020
 ---
 
-{{< lead >}}
-Investigate context-dependent aggression triggered by frustrative
-non-reward or acute social threats. Using newly developed approaches, multiple behavioral domains will be assessed in
-a semi-naturalistic, autonomous mouse habitat. Specifically, the habitat assesses the inter-individual dynamics of social
-interactions, aggressions, and hierarchy and the individual reward learning and impulsivity through different integrated
-modules. Intermittent challenges comprise intruder aggression and frustrative non-rewards. Within this LCD, circuit
-mechanisms are dissected through chemogenetic interventions, in vivo recordings, and functional MRI in awake mice
-during task performance. This approach in the first funding period will enable us to disentangle the specific functions of
-candidate entry points in prefrontal to ventral striatum pathways with respect to their modulation of aggression and
-dominance for potential interventions.
-{{< /lead >}}
+Investigate context-dependent aggression triggered by frustrative non-reward or
+acute social threats. Using newly developed approaches, multiple behavioral
+domains will be assessed in a semi-naturalistic, autonomous mouse habitat.
+Specifically, the habitat assesses the inter-individual dynamics of social
+interactions, aggressions, and hierarchy and the individual reward learning and
+impulsivity through different integrated modules. Intermittent challenges
+comprise intruder aggression and frustrative non-rewards. Within this LCD,
+circuit mechanisms are dissected through chemogenetic interventions, in vivo
+recordings, and functional MRI in awake mice during task performance. This
+approach in the first funding period will enable us to disentangle the specific
+functions of candidate entry points in prefrontal to ventral striatum pathways
+with respect to their modulation of aggression and dominance for potential
+interventions.

content/projects/c04/_index.md

@@ -1,18 +1,33 @@
 ---
-title: "C04: The sex-specific role of genes, early adversity, peers, community violence, and puberty related endocrinological changes in adolescent pathological aggression"
+title: 'C04: The sex-specific role of genes, early adversity, peers, community violence,
+  and puberty related endocrinological changes in adolescent pathological aggression'
 contributors:
-- Christine Margarete Freitag
-- Andreas G Chiocchetti
+- andreas-g-chiocchetti
+- christine-margarete-freitag
+- moritz-sturm
+- simeon-platte
 sites:
 - frankfurt
 topics:
-- research-area-c
 - dfg-206-09
+- research-area-c
 - dfg-206-10
 weight: 3030
 ---
 
-{{< lead >}}
-Address sex-specific NVS (reactive aggression) and CS (different dimensions of psychopathy, proactive aggression) associated risk factors, and risk factor-based biosignatures in young people. Considering the interacting genetic, environmental, and hormonal factors related to these specific aggressive behavior dimensions, C04 will identify specific and shared factors and mechanisms related to
-NVS and CS in female and male youth with and without pathological aggression. Implementing deep-learning algorithms, sex-specific, data-driven subgroups in relation to dimensions of aggressive behavior will be described and probed against the NVS and CS. Group-level risk factors of aggressive behavior dimensions, and individual risk factor-based subgrouping will be the basis of developing a biologically informed stratification strategy for tailored treatment. Models and classifiers will be established cross-sectionally in available data and replicated in the prospectively collected cross-sectional data (Q01). In addition, C04 will test the models and classifiers for predictive validity in the longitudinal data of the TRR Q01 cohort.
-{{< /lead >}}
+Address sex-specific NVS (reactive aggression) and CS (different dimensions of
+psychopathy, proactive aggression) associated risk factors, and risk factor-
+based biosignatures in young people. Considering the interacting genetic,
+environmental, and hormonal factors related to these specific aggressive
+behavior dimensions, C04 will identify specific and shared factors and
+mechanisms related to NVS and CS in female and male youth with and without
+pathological aggression. Implementing deep-learning algorithms, sex-specific,
+data-driven subgroups in relation to dimensions of aggressive behavior will be
+described and probed against the NVS and CS. Group-level risk factors of
+aggressive behavior dimensions, and individual risk factor-based subgrouping
+will be the basis of developing a biologically informed stratification strategy
+for tailored treatment. Models and classifiers will be established cross-
+sectionally in available data and replicated in the prospectively collected
+cross-sectional data (Q01). In addition, C04 will test the models and
+classifiers for predictive validity in the longitudinal data of the TRR Q01
+cohort.

content/projects/c05/_index.md

@@ -1,20 +1,23 @@
 ---
-title: "C05: The neuroanatomical underpinnings of clinical aggression and their relationship with the negative valence and cognitive control systems"
+title: 'C05: The neuroanatomical underpinnings of clinical aggression and their relationship
+  with the negative valence and cognitive control systems'
 contributors:
-- Christine Ecker
+- alessio-giacomel
+- christine-ecker
+- wiebke-hennig
 sites:
 - frankfurt
 topics:
-- research-area-c
 - dfg-206-09
+- research-area-c
 weight: 3040
 ---
 
-{{< lead >}}
-Link questionnaire measures of aggression to specific neural
-substrates using structural MRI. The resulting patterns of aggression-related neuroanatomical variability will be co-
-registered with the Allen Human Brain Atlas providing gene-expression data, to highlight genes with a spatial pattern
-of expression that matches the neuroimaging findings. Utilizing the neurotypical control data, a normative model of
-neuroanatomical diversity within the NVS and CS will be established to quantify neuroanatomical abnormalities within
-these systems in individual cases
-{{< /lead >}}
+Link questionnaire measures of aggression to specific neural substrates using
+structural MRI. The resulting patterns of aggression-related neuroanatomical
+variability will be co- registered with the Allen Human Brain Atlas providing
+gene-expression data, to highlight genes with a spatial pattern of expression
+that matches the neuroimaging findings. Utilizing the neurotypical control data,
+a normative model of neuroanatomical diversity within the NVS and CS will be
+established to quantify neuroanatomical abnormalities within these systems in
+individual cases.

content/projects/c06/_index.md

@@ -1,8 +1,9 @@
 ---
-title: "C06: Brain mechanisms differentiating aggressive vs. non-aggressive psychopathology as sequelae of early life maltreatment"
+title: 'C06: Brain mechanisms differentiating aggressive vs. non-aggressive psychopathology
+  as sequelae of early life maltreatment'
 contributors:
-- Sabine Herpertz
-- Corinne Neukel
+- corinne-neukel
+- sabine-herpertz
 sites:
 - heidelberg
 topics:
@@ -11,13 +12,14 @@ topics:
 weight: 3050
 ---
 
-{{< lead >}}
-Identify specific neuronal mechanisms related to the NVS
-and CS in female and male clinical samples with a history of early-life maltreatment (ELM) who exhibit externalizing,
-aggressive psychopathologies as opposed to internalizing, non-aggressive psychopathologies. We will therefore explore
-the interaction of the NVS and CS as well as the modulating effects of theory-of-mind (ToM) on the NVS and CS using a
-series of fMRI and behavioral tasks. Furthermore, we will investigate the role of hormonal stress responses and will
-use EMA to assess anger and aggression in everyday life. Thus, we will be able to combine behavioral phenotyping in
-natural conditions of everyday life and neurobiological correlates of psychopathology in order to detect clinically relevant
-biosignatures for AMD.
-{{< /lead >}}
+Identify specific neuronal mechanisms related to the NVS and CS in female and
+male clinical samples with a history of early-life maltreatment (ELM) who
+exhibit externalizing, aggressive psychopathologies as opposed to internalizing,
+non-aggressive psychopathologies. We will therefore explore the interaction of
+the NVS and CS as well as the modulating effects of theory-of-mind (ToM) on the
+NVS and CS using a series of fMRI and behavioral tasks. Furthermore, we will
+investigate the role of hormonal stress responses and will use EMA to assess
+anger and aggression in everyday life. Thus, we will be able to combine
+behavioral phenotyping in natural conditions of everyday life and
+neurobiological correlates of psychopathology in order to detect clinically
+relevant biosignatures for AMD.

content/projects/c07/_index.md

@@ -1,22 +1,24 @@
 ---
-title: "C07:  Identifying mediators of threat-aggression and experimental manipulation by tDCS"
+title: 'C07: Identifying mediators of threat-aggression and experimental manipulation
+  by tDCS'
 contributors:
-- Michael Plichta
-- Wolfgang Retz
+- michael-plichta
+- wolfgang-retz
 sites:
 - frankfurt
 - mainz
 topics:
-- research-area-c
 - dfg-206-09
+- research-area-c
 weight: 3060
 ---
 
-{{< lead >}}
-Test the interaction of the CS and frustrative non-reward as part of the NVS. It will investigate the electrophysiological
-correlates of frustrative feedback in aggression-prone patients. In the aftermath of induced stress, an EEG task-battery
-including frustrative feedback will be applied for extraction of error-related negativity (ERN) and contingent negative
-variation to monitor electro-physiologic signaling of the relevant learning and frustration processes. In half of the
-participants, tDCS over the prefrontal cortex will be applied to enhance cognitive control, with participants being put into
-a stress context inducing frustration.
-{{< /lead >}}
+Test the interaction of the CS and frustrative non-reward as part of the NVS. It
+will investigate the electrophysiological correlates of frustrative feedback in
+aggression-prone patients. In the aftermath of induced stress, an EEG task-
+battery including frustrative feedback will be applied for extraction of error-
+related negativity (ERN) and contingent negative variation to monitor electro-
+physiologic signaling of the relevant learning and frustration processes. In
+half of the participants, tDCS over the prefrontal cortex will be applied to
+enhance cognitive control, with participants being put into a stress context
+inducing frustration.

content/projects/q01/_index.md

@@ -1,21 +1,32 @@
 ---
-title: "Q01: Recruitment and biotyping transdiagnostic risk mechanisms for aggressive behaviors in mental disorders across the life span"
+title: 'Q01: Recruitment and biotyping transdiagnostic risk mechanisms for aggressive
+  behaviors in mental disorders across the life span'
 contributors:
-- Tobias Banaschewski
-- Thomas Frodl
-- Sabine Herpertz
-- Andreas Reif
-- Christine Margarete Freitag
-- Ute Habel
-- Kerstin Konrad
-- Christiane Licht
-- Christina Neczewicz
-- Celina Müller
+- andreas-reif
+- annika-maas
+- antonia-fritsch
+- catherine-barnes-scheufler
+- celina-mueller
+- christiane-licht
+- christina-neczewicz
+- christine-margarete-freitag
+- dario-mueller
+- henry-schirok
+- ina-kuschel
+- jaqueline-scharf
+- kerstin-konrad
+- philippa-huepen
+- robert-kraemer
+- sabine-herpertz
+- thilo-kellermann
+- thomas-frodl
+- tobias-banaschewski
+- ute-habel
 sites:
 - aachen
-- mannheim
-- heidelberg
 - frankfurt
+- heidelberg
+- mannheim
 topics:
 - dfg-206-09
 - dfg-206-10
@@ -24,8 +35,7 @@ roles:
 weight: 4000
 ---
 
-{{< lead >}}
-The central recruitment platform for collecting and curating a longitudinal dataset for studying individual aggression
-dynamics related to the neural, cognitive-emotional, neurobiological, psychopathological and environmental factors in
-patient groups.
-{{< /lead >}}
+The central recruitment platform for collecting and curating a longitudinal
+dataset for studying individual aggression dynamics related to the neural,
+cognitive-emotional, neurobiological, psychopathological and environmental
+factors in patient groups.

content/projects/q02/_index.md

@@ -1,38 +1,36 @@
 ---
-title: "Q02: Data management for computational modelling"
+title: 'Q02: Data management for computational modelling'
 contributors:
-- Michael Hanke
-- Gabriele Ende
-- Richard Nkrumah
-- Christine Ecker
-- Klaus Mathiak
-- Wiebke Hennig
-- Arezoo Taebi
+- alessio-giacomel
+- arezoo-taebi
+- christine-ecker
+- gabriele-ende
+- klaus-mathiak
+- michael-hanke
+- richard-nkrumah
+- stephan-heunis
+- traute-demirakca
+- wiebke-hennig
 sites:
+- aachen
+- frankfurt
 - juelich
 - mannheim
-- frankfurt
-- aachen
 roles:
 - service-project
 weight: 4010
 ---
 
-{{< lead >}}
 Data management and training platform. A decentralized data management
 infrastructure will help focus on developmental and therapeutic longitudinal
 data, training all participating researchers in the necessary skills for future
 use. This strategy will lay the foundations for further data-driven
-computational modelling projects in the next funding period.
-{{< /lead >}}
-
-This is a distributed project, with representatives at all main TRR379 sites.
-
-As a key software solution, this project employs [DataLad](https://datalad.org).
-DataLad is a data management software designed to facilitate the organization,
-sharing, and reproducibility of scientific datasets. It integrates version
-control with data handling, allowing researchers to track changes, collaborate
-efficiently, and ensure the accessibility and integrity of their data. By
-leveraging tools like Git and Git-annex, DataLad provides a streamlined way to
-manage large datasets, making it particularly valuable in fields like
-neuroimaging and bioinformatics.
+computational modelling projects in the next funding period. This is a
+distributed project, with representatives at all main TRR379 sites. As a key
+software solution, this project employs DataLad. DataLad is a data management
+software designed to facilitate the organization, sharing, and reproducibility
+of scientific datasets. It integrates version control with data handling,
+allowing researchers to track changes, collaborate efficiently, and ensure the
+accessibility and integrity of their data. By leveraging tools like Git and Git-
+annex, DataLad provides a streamlined way to manage large datasets, making it
+particularly valuable in fields like neuroimaging and bioinformatics.

content/projects/q03/_index.md

@@ -1,19 +1,17 @@
 ---
-title: "Q03: Integrated Research Training Group (RTG)"
+title: 'Q03: Integrated Research Training Group (RTG)'
 contributors:
-- David Slattery
-- Lisa Wagels
-- Tobias Banaschewski
+- david-slattery
+- lisa-wagels
+- tobias-banaschewski
 sites:
-- frankfurt
 - aachen
+- frankfurt
 - mannheim
 roles:
 - service-project
 weight: 4020
 ---
 
-{{< lead >}}
-Bundle all training and mentoring activities in a research training
-group (RTG) for the young researchers participating in this collaborative research center.
-{{< /lead >}}
+Bundle all training and mentoring activities in a research training group (RTG)
+for the young researchers participating in this collaborative research center.

content/projects/q04/_index.md

@@ -1,7 +1,12 @@
 ---
-title: "Q04: Central coordination"
+title: 'Q04: Central coordination'
 contributors:
-- Ute Habel
+- anja-conzen
+- anna-kwiatkowski
+- oliver-guenther
+- philippa-huepen
+- ute-habel
+- volker-backes
 sites:
 - aachen
 roles:
@@ -9,6 +14,4 @@ roles:
 weight: 4030
 ---
 
-{{< lead >}}
 Governance, and communication among participating researchers and the public.
-{{< /lead >}}